Peter Kovermann Selected Research

Type 6 Episodic Ataxia

12/2013	Mutating a conserved proline residue within the trimerization domain modifies Na+ binding to excitatory amino acid transporters and associated conformational changes.
11/2012	A point mutation associated with episodic ataxia 6 increases glutamate transporter anion currents.

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Peter Kovermann Research Topics

Disease

2	Hemiplegia (Spastic Hemiplegia) 01/2020 - 10/2017
2	Episodic Ataxia 01/2020 - 11/2012
2	Type 6 Episodic Ataxia 12/2013 - 11/2012
1	Brain Diseases (Brain Disorder) 01/2022
1	Inborn Genetic Diseases (Disease, Hereditary) 01/2021
1	Traumatic Brain Injuries (Traumatic Brain Injury) 01/2021
1	Infections 01/2021
1	Ischemia 01/2021
1	Ataxia (Dyssynergia) 01/2020
1	Epilepsy (Aura) 01/2020
1	Migraine Disorders (Migraine) 10/2017
1	Migraine with Aura (Familial Hemiplegic Migraine) 10/2017

Drug/Important Bio-Agent (IBA)

5	AnionsIBA 01/2022 - 11/2012
3	Proline (L-Proline)FDA Link 01/2020 - 12/2013
3	Amino Acid Transport System X-AG (Glutamate Transporter)IBA 01/2020 - 11/2012
2	Arginine (L-Arginine)FDA Link 01/2020 - 12/2013
2	Glutamic Acid (Glutamate)FDA Link 12/2013 - 11/2012
1	Excitatory Amino Acid Transporter 2IBA 01/2022
1	ChloridesIBA 01/2021
1	Excitatory Amino Acid Transporter 1 (Glutamate Aspartate Transporter 1)IBA 01/2020
1	Threonine (L-Threonine)FDA Link 10/2017
1	Ion Channels (Ion Channel)IBA 11/2012
1	Excitatory Amino AcidsIBA 11/2012